Search results for "Genome Stability"

showing 7 items of 7 documents

Impact of DLK1-DIO3 imprinted cluster hypomethylation in smoker patients with lung cancer.

2018

// Sonia Molina-Pinelo 1, 2 , Ana Salinas 1 , Nicolas Moreno-Mata 2 , Irene Ferrer 1, 2 , Rocio Suarez 1, 2 , Eduardo Andres-Leon 1 , Manuel Rodriguez-Paredes 4, 5 , Julian Gutekunst 4 , Eloisa Jantus-Lewintre 6, 7 , Carlos Camps 8, 9 , Amancio Carnero 1 , Luis Paz-Ares 1, 2 1 Instituto de Biomedicina de Sevilla (IBIS) (HUVR, CSIC, Universidad de Sevilla), Sevilla, Spain 2 Medical Oncology Department, Hospital Universitario Doce de Octubre & Centro Nacional de Investigaciones Oncologicas (CNIO), Madrid, Spain 3 Thoracic Surgery Department, Hospital Universitario Virgen del Rocio, Sevilla, Spain 4 Division of Epigenetics, DKFZ-ZMBH Alliance, German Cancer Research Center, Heidelberg, Germany…

0301 basic medicineOncologymedicine.medical_specialtyPathology03 medical and health sciences0302 clinical medicineInternal medicineCancer genomeMedicineCOPDIn patienttranscriptional regulationLung cancerGenome stabilityThoracic surgery departmentbusiness.industryDLK1-DIO3 clustermedicine.diseaselung cancer030104 developmental biologyOncologyNormal lung030220 oncology & carcinogenesisDNA methylationbusinessLung tissueepigeneticResearch Paper
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Hypoxia and Human Genome Stability: Downregulation of BRCA2 Expression in Breast Cancer Cell Lines

2013

Previously, it has been reported that hypoxia causes increased mutagenesis and alteration in DNA repair mechanisms. In 2005, an interesting study showed that hypoxia-induced decreases in BRCA1 expression and the consequent suppression of homologous recombination may lead to genetic instability. However, nothing is yet known about the involvement of BRCA2 in hypoxic conditions in breast cancer. Initially, a cell proliferation assay allowed us to hypothesize that hypoxia could negatively regulate the breast cancer cell growth in short term in vitro studies. Subsequently, we analyzed gene expression in breast cancer cell lines exposed to hypoxic condition by microarray analysis. Interestingly,…

Genome instabilityDNA RepairArticle SubjectDNA repairDNA damageSettore MED/06 - Oncologia MedicaDown-Regulationlcsh:MedicineBreast NeoplasmsBiologyGeneral Biochemistry Genetics and Molecular BiologyGenomic InstabilityBreast cancerCell Line TumorBreast CancermedicineHumansEnzyme Inhibitorsskin and connective tissue diseasesHypoxiaBiologyGeneral Immunology and MicrobiologyBRCA1 ProteinGenome Humanlcsh:RGenome StabilityGeneral MedicineDNA repair protein XRCC4medicine.diseaseBRCA2Cell HypoxiaAmino Acids DicarboxylicGene Expression Regulation NeoplasticCancer researchDNA mismatch repairFemaleHuman medicineHypoxia; Genome Stability; BRCA2; Breast CancerHomologous recombinationEngineering sciences. TechnologyNucleotide excision repairResearch ArticleDNA Damage
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Between Scylla and Charibdis: eIF2α kinases as targets for cancer chemotherapy

2011

[EN] The eIF2 alpha kinases integrate translation initiation rates with nutrient availability, thus allowing cells to adapt to nutrient scarcity. Recent evidence has uncovered new functions of these kinases in tumour cell biology, ranging from regulation of cell cycle progression, maintenance of genome stability, control of apoptosis, and cell survival under nutrient stress and hypoxia. Accordingly, active eIF2 alpha kinases modulate the antineoplasic activity of several antitumour drugs, either by exacerbating their cytotoxic effect or by promoting chemoresistance. Understanding of eIF2 alpha kinases molecular roles may provide mechanistic insights into how tumour cells sense and adapt to …

PERKBioquímicaTranslationBiologiaCancer ResearchCancer chemotherapyEukaryotic Initiation Factor-2Antineoplastic AgentsBiologyBioinformaticsNeoplasmsBIOQUIMICA Y BIOLOGIA MOLECULARHumansCytotoxic T cellCell survivalGenome stabilityKinaseNutrient stressPKRGeneral MedicineProtein kinase ROncologyApoptosiseIF2 alpha phosphorylationCancer researchGCN2Clinical and Translational Oncology
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Experimental evolution of genome architecture and complexity in an RNA virus

2016

Introducción La evolución de la arquitectura del genoma – las dimensiones y la organización del material hereditario de un organismo – es poco conocida. Existe una variación asombrosa en la arquitectura genómica entre diferentes organismos. Los virus tienden a tener genomas pequeños, con un mínimo de secuencias intergénicas y típicamente con genes solapantes, los cuales se cree son una forma de compresión del genoma que permite al virus incrementar su número de proteínas sin aumentar su tamaño. Los procariotas tienen genomas compactos, pero con secuencias intergénicas más largas que los virus, y los genes con solapamientos largos son escasos. Los eucariotas presentan una amplia gama de tama…

virus evolutiongenome architectureRNA virusUNESCO::CIENCIAS DE LA VIDAexperimental evolutiongenome evolutiongenome stability:CIENCIAS DE LA VIDA [UNESCO]
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Role of the Non-Canonical RNAi Pathway in the Antifungal Resistance and Virulence of Mucorales

2021

Mucorales are the causal agents for the lethal disease known as mucormycosis. Mortality rates of mucormycosis can reach up to 90%, due to the mucoralean antifungal drug resistance and the lack of effective therapies. A concerning urgency among the medical and scientific community claims to find targets for the development of new treatments. Here, we reviewed different studies describing the role and machinery of a novel non-canonical RNAi pathway (NCRIP) only conserved in Mucorales. Its non-canonical features are the independence of Dicer and Argonaute proteins. Conversely, NCRIP relies on RNA-dependent RNA Polymerases (RdRP) and an atypical ribonuclease III (RNase III). NCRIP regulates the…

AntifungalTransposable element0301 basic medicineMucoralesAntifungal Agentstransposonmedicine.drug_classRNA Stability030106 microbiologyAntifungal drugVirulenceReviewQH426-470mucormycosis03 medical and health sciencesDrug Resistance FungalRNA interferenceFongsmedicineGeneticsbiochemistryRNA MessengerRibonuclease IIIepimutantGenetics (clinical)Genome stabilityGeneticsRdRPR3B2biologyMucormycosisnon-canonical RNAiRNA FungalArgonauteantifungal resistancemedicine.diseasebiology.organism_classificationvirulenceRNA silencing030104 developmental biologyNon canonicalbiology.proteinInfeccióMucoralesRNA Interferencegenome stabilitySignal TransductionDicerGenes
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Analysis of DNA Polymerases Reveals Specific Genes Expansion in Leishmania and Trypanosoma spp.

2020

Leishmaniasis and trypanosomiasis are largely neglected diseases prevailing in tropical and subtropical conditions. These are an arthropod-borne zoonosis that affects humans and some animals and is caused by infection with protozoan of the genera Leishmania and Trypanosoma, respectively. These parasites present high genomic plasticity and are able to adapt themselves to adverse conditions like the attack of host cells or toxicity induced by drug exposure. Different mechanisms allow these adapting responses induced by stress, such as mutation, chromosomal rearrangements, establishment of mosaic ploidies, and gene expansion. Here we describe how a subset of genes encoding for DNA polymerases …

0301 basic medicineMicrobiology (medical)TrypanosomaDNA polymeraseDNA repairgene amplification030106 microbiologyImmunologylcsh:QR1-502DNA repairtrypanosomatidsDNA-Directed DNA Polymerasemedicine.disease_causeMicrobiologylcsh:Microbiology03 medical and health sciencesDNA polymerasesCellular and Infection MicrobiologyTrypanosomiasisGene duplicationTrypanosomatidamedicineAnimalsHumanstranslesion synthesisGeneLeishmaniasisGeneticsLeishmaniaMutationbiologyLeishmaniabiology.organism_classification030104 developmental biologyInfectious DiseasesPerspectivebiology.proteinTrypanosomagenome stabilityFrontiers in Cellular and Infection Microbiology
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Genome Stability in Embryonic Stem Cells

2011

Paola Rebuzzini1, Maurizio Zuccotti2*, Carlo Alberto Redi1,3 and Silvia Garagna1,4,5* 1Laboratorio di Biologia dello Sviluppo, Dipartimento di Biologia Animale, Universita degli Studi di Pavia, Via Ferrata 9, 27100 Pavia, 2Sezione di Istologia ed Embriologia, Dipartimento di Medicina Sperimentale, Universita degli Studi di Parma, Via Volturno 39, 43100 Parma 3Fondazione I.R.C.C.S. Policlinico San Matteo, Piazzale Golgi, 19, 27100 Pavia 4Centro di Ricerca Interdipartimentale di Ingegneria Tissutale, Universita degli Studi di Pavia, Via Ferrata 1, 27100 Pavia 5Centro di Eccellenza in Biologia Applicata, Universita degli Studi di Pavia, Via Ferrata 9, 27100 Pavia Italy

Homeobox protein NANOGCellular differentiationRex1Embryoid bodyStem cellBiologyInduced pluripotent stem cellMolecular biologyEmbryonic stem cellGenome stability
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